moon blindness - definizione. Che cos'è moon blindness
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Cosa (chi) è moon blindness - definizione

CONDITION MAKING IT DIFFICULT OR IMPOSSIBLE TO SEE IN RELATIVELY LOW LIGHT
Night-blindness; Night blindness; Night Blindness; Moon blink; Moonblink; Night blind; Nightblindness; Nightblind; Cyntolopia; Nyctalopic
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Equine recurrent uveitis         
  • Advanced stage of disease
  • Advanced stage of disease
  • ERU on an [[Icelandic horse]]
EYE DISEASE OF HORSES
Moon blindness; Equine Recurrent Uveitis
Equine recurrent uveitis (ERU) – also known as moon blindness, recurrent iridocyclitis, or periodic ophthalmia – is an acute, nongranulomatous inflammation of the uveal tract of the eye, occurring commonly in horses of all breeds, worldwide. The causative factor is not known, but several pathogeneses have been suggested.
Nyctalopia         
·noun ·see Moonblink.
II. Nyctalopia ·noun A disease of the eye, in consequence of which the patient can see well in a faint light or at twilight, but is unable to see during the day or in a strong light; day blindness.
nyctalopia         
[?n?kt?'l??p??]
¦ noun a condition characterized by an abnormal inability to see in dim light or at night, typically caused by vitamin A deficiency.
Origin
C17: via late L. from Gk nuktalops, from nux, nukt- 'night' + alaos 'blind' + ops 'eye'.

Wikipedia

Nyctalopia

Nyctalopia (; from Ancient Greek νύκτ- (núkt-) 'night', ἀλαός (alaós) 'blind, invisible', and ὄψ (óps) 'eye'), also called night-blindness, is a condition making it difficult or impossible to see in relatively low light. It is a symptom of several eye diseases. Night blindness may exist from birth, or be caused by injury or malnutrition (for example, vitamin A deficiency). It can be described as insufficient adaptation to darkness.

The most common cause of nyctalopia is retinitis pigmentosa, a disorder in which the rod cells in the retina gradually lose their ability to respond to the light. Patients with this genetic condition have progressive nyctalopia and eventually, their daytime vision may also be affected. In X-linked congenital stationary night blindness, from birth the rods either do not work at all, or work very little, but the condition does not get worse.

Another cause of night blindness is a deficiency of retinol, or vitamin A1, found in fish oils, liver and dairy products.

The opposite problem, the inability to see in bright light, is known as hemeralopia and is much rarer.

Since the outer area of the retina is made up of more rods than cones, loss of peripheral vision often results in night blindness. Individuals with night blindness not only see poorly at night but also require extra time for their eyes to adjust from brightly lit areas to dim ones. Contrast vision may also be greatly reduced.

Rods contain a receptor-protein called rhodopsin. When light falls on rhodopsin, it undergoes a series of conformational changes ultimately generating electrical signals which are carried to the brain via the optic nerve. In the absence of light, rhodopsin is regenerated. The body synthesizes rhodopsin from vitamin A, which is why a deficiency in vitamin A causes poor night vision.

Refractive "vision correction" surgery (especially PRK with the complication of "haze") may rarely cause a reduction in best night-time acuity due to the impairment of contrast sensitivity function (CSF) which is induced by intraocular light-scatter resulting from surgical intervention in the natural structural integrity of the cornea.